In ferrimagnetic GdFeCo, when the temperature approaches the angular momentum compensation point T(A), both frequency of FMR mode and the Gilbert damping parameter increase significantly. High-speed and strongly damped precessional switching was triggered with ultrafast heating of a Gd(24.5)Fe(66.1)Co(9.4) across its magnetization compensation point T(M) up to T(A), under a static applied magnetic field. To initiate and investigate

the magnetization reversal, we have used an all-optical pump-probe technique employing an amplified Ti: sapphire laser system with 90 fs pulses. In particular, following the laser excitation with low pump fluence (0.9 mJ/cm(2)) the magnetic system relaxes back toward initial state while the rather high pump fluence (3.3 mJ/cm(2)) excitation induces a metastable opposite www.selleckchem.com/products/epz-5676.html magnetic state. After the sudden heating which causes just 30% reduction of magnetization at first breakdown, consequently magnetization was started to rotate across M(z) = 0 within 6 ps during first precession and finished the high damped precessional motion within few cycles into opposite direction. (C) 2011 American Institute of Physics. [doi: 10.1063/1.3535415]“
“Because in the early phases of spinal cord ischemia magnetic resonance imaging (MRI) can be normal, its clinical diagnosis is often difficult. We aimed to explore if motor-evoked potentials

(MEPs) recordings may contribute to earlier diagnosis of spinal cord stroke. The clinical, MRI, and MEP findings in one case each of cervical and GDC-0068 inhibitor lumbar spinal cord infarction were reported. Spinal MRI at admission was unremarkable in both patients. At this time, MEPs were abnormal in both patients, to the upper and lower limbs in the first patient, exclusively to the lower limbs in the second. Follow-up MRI examinations documented an infarction in the territory of the anterior spinal artery and of the Adamkiewicz artery, respectively. MEP study can be useful in demonstrating spinal cord involvement also when radiological evidence

for spinal cord damage is absent or equivocal. Early diagnosis may allow earlier intervention and contribute to improved patient management.”
“Hepatitis C virus (HCV) causes a chronic liver infection, which may IWR-1-endo result in cirrhosis and hepatocellular carcinoma. Impairment of the maturation process in dendritic cells (DCs) may be one of the mechanisms responsible for immune evasion of HCV. The core and NS3 proteins are among the most conserved HCV proteins and play a key role in viral clearance. To evaluate the effects of these proteins on DCs, monocyte-derived immature DCs (iDCs) were transfected with in vitro transcribed (IVT) HCV core or NS3 RNA and treated with maturation factors. Neither core nor NS3 had an inhibitory effect on DC maturation; however, transfection of iDCs with IVT core RNA appeared to result in changes compatible with maturation.